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  • 1
    ISSN: 1432-1238
    Keywords: Severe cerebral trauma ; Midbrain syndrome ; Apallic syndrome ; Metabolic rates ; Catabolism ; High caloric nutrition
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Summary 27 Patients following severe brain injury were studied with regard to metabolic alterations. Metabolic rate was calculated from measured RQ, respiratory minute volume and oxygen consumption, as soon as the patient was able to breathe spontaneously. Catabolism was defined by determination of N-balance. It was concluded that metabolism following cerebral injury is particularly enhanced compared to common trauma. The neurological course of patients, who after midbrain syndrome recovered in contrast to those who developed apallic syndrome had little influence on metabolic rates, however, it distinctly seemed to modify catabolism. Thus catabolism throughout the apallic phase was found to be higher. The requirements for a favourable recovery from severe brain injury were defined as a well balanced nitrogen regime apart from a high amount of carbohydrate and fat calories.
    Type of Medium: Electronic Resource
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  • 2
    ISSN: 1432-1238
    Keywords: Severe cerebral trauma ; Midbrain syndrome ; Apallic syndrome ; Catecholamines ; Fat oxidation ; Thyroid hormones ; High caloric total parenteral alimentation (TPA)
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Abstract Urinary catecholamine excretion and thyroid hormone blood level were studied in 16 patients following severe cerebral trauma. Increased excretion rates of epinephrine and norepinephrine were found. There was no significant difference in the catecholamine excretion when compared with generally traumatized patients. The relationships between catecholamine excretion, increased metabolic rates, and negative nitrogen balance indicate that in patients with a midbrain syndrome there exists an additional diencephalic metabolic factor, which leads to a rise in fat oxidation and perpetuation of catabolism. Early high caloric parenteral nutrition seems to inhibit the initial increase of catecholamine excretion and thus protects the body from an unnecessary breakdown of its own reserves. If the course is classified according to neurological stages, it can be shown that patients with a traumatic apallic syndrome in poor condition have a high increase of catecholamine excretion. Secretion of thyroid hormones is not influenced significantly by cerebral trauma.
    Type of Medium: Electronic Resource
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