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  • 1
    Keywords: Medicine ; Toxicology ; Pharmaceutical technology ; Chemistry, Organic ; Chemical engineering ; Biochemistry ; Biomedicine ; Pharmacology/Toxicology ; Pharmaceutical Sciences/Technology ; Organic Chemistry ; Medicinal Chemistry ; Medical Biochemistry ; Industrial Chemistry/Chemical Engineering ; Springer eBooks
    Pages: : digital
    ISBN: 9783034801256
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  • 2
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    Basel : Birkhäuser
    Keywords: Medicine ; Immunology ; Toxicology ; Allergy ; Emerging infectious diseases ; Biochemistry ; Cytology ; Biomedicine ; Immunology ; Pharmacology/Toxicology ; Cell Biology ; Infectious Diseases ; Allergology ; Medical Biochemistry ; Springer eBooks
    Pages: : digital
    Edition: 3rd revised and extended edition
    ISBN: 9783034601368
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  • 3
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    Basel : Springer
    Keywords: Medicine ; Immunology ; Pharmacology ; Infectious Diseases ; Rheumatology ; Cell Biology ; Biomedicine ; Immunology ; Pharmacology/Toxicology ; Infectious Diseases ; Cell Biology ; Rheumatology ; Springer eBooks
    Description / Table of Contents: Adhesion molecules of neutrophils -- Anaphylaxis (Immediate hypersensitivity): from old to new mechanisms -- Antiviral Responses -- Autophagy and Inflammation -- Basophils -- Cell signalling in neutrophils -- Co-stimulatory Receptors -- Dendritic Cells -- GILZ-Related Regulation of Inflammation -- Glial Cells -- Immunoglobulin receptors and inflammation.-Inflammation and autoimmunity -- Inflammatory Bowel Disease Models in Animals -- Leukocyte Recruitment -- Lymphocyte homing and trafficking -- Macrophage heterogeneity during inflammation -- Mast Cells -- Mechanisms of Macrophage Migration in 3-Dimensional Environments -- Microvascular Responses to Inflammation -- Modulation of inflammation by key nutrials -- Natural Killer Cells -- Neutrophil Extracellular Traps -- Neutrophil oxidative burst -- Obesity and Inflammation -- Osteoclasts in Inflammation -- Parasitism: A Unique Lifestyle Reflecting Dynamic Parasite- and Host(s)-Co-driven Processes -- Platelet, Endothelium, and Inflammation -- Regulatory T cells -- Sepsis Models in Animals -- Skin inflammation models in animals -- Th17 Response -- Toll-Like Receptors -- Angiogenesis Inhibitors -- Anti-asthma Drugs, Overview -- Anti-histamines -- Anti-muscarinics -- Antibiotics as anti-inflammatory drugs -- Antimalarial Drugs -- Azathioprine/Thiopurines -- Beta2 Agonists -- CD 20 Inhibitors – Rituximab -- CD80/86 Inhibitors -- Chromones – Cromoglycate and Nedocromil -- Colony stimulating factor therapeutics -- Corticosteroids -- Coxibs -- Disease-Modifying Anti-Rheumatic Drugs -- Fenamates -- Flavonoids as anti-inflammatory agents -- Gold Complexes -- Interleukin 1 Inhibitors -- Interleukin 12/23 Inhibitors: Ustekinumab Interleukin-6 Inhibitor – Tocilizumab -- Leflunomide -- Medicinal Fatty Acids -- Methotrexate.-NO-donating anti-inflammatory drugs -- Non-steroidal anti-inflammatory drugs -- Phosphodiesterase-4 inhibitors -- Propionic Acid Derivative Drugs (Profens) -- Salicylates -- Sulfasalazine and Related Drugs -- Tetracyclines -- Theophylline -- TNF Inhibitors -- Acute exacerbations of airway inflammation -- Allergic Disorders -- Alzheimer's Disease -- Antiphospholipid antibody syndrome -- Asthma -- Atopic dermatitis -- Autoinflammatory syndromes -- Behcet’s disease -- Bronchiolitis obliterans -- Cancer and inflammation -- Cogan’s syndrome -- COPD -- Diabetes type I -- Diabetes type II -- Diffuse panbronchiolitis -- Gaucher's disease -- Genetic susceptibility to inflammatory diseases -- Gout -- Graft vs Host Disease -- Granulomatosis with Polyangiitis (GPA) -- Grave’s disease.-Guillaine-Barre disease -- Henoch-Schönlein Purpura -- Idiopathic Thrombocytopenic Purpura -- Inflammatory bowel disease -- Juvenile idiopathic arthritis -- Kawasaki Disease -- Lyme disease -- Osteoarthritis.-Osteoarthritis Genetics -- Polymyositis and Dermatomyositis -- Rheumatic Fever -- Rheumatoid arthritis -- Role of Cell Therapy in Autoimmune Disease -- Sarcoidosis -- Sepsis -- Spondylarthropathies -- ACKR3 -- Adhesion molecules -- Alarmins -- Antibacterial Host .-Defense Peptides -- Bacterial Lipopolysaccharide.-Biofilm -- Bone Morphogenetic Proteins in Inflammation -- C5a Receptors -- CCL18 -- CCR1 -- Ceramide 1-Phosphate: A Mediator of Inflammatory Responses -- Complement System -- CXCL4 und CXCL4L1 -- CXCR1 and CXCR2 and Ligands -- CXCR3 and its Ligands -- Cytokines -- Cytolytic Granules -- Foxp3 -- Heat Shock Proteins -- Histamine -- IFNgamma -- IkappaB -- Inflammasomes -- Interleukin 1 -- Interleukin 10 -- Interleukin 12 -- Interleukin 17 -- Interleukin 18 -- Interleukin 18 Binding Protein -- Interleukin 2 -- Interleukin 22 -- Interleukin 23 -- Interleukin 27 -- Interleukin 32 -- Interleukin 36 Cytokines -- Interleukin 4 and the related cytokines (Interleukin 5 and Interleukin 13) -- Interleukin 6 -- Interleukin 9 -- Interleukin-33 -- Janus Kinases (JAKs)/STAT Pathway -- Kinins -- Leukotrienes -- Mammalian Target of Rapamycin (mTOR) -- MAP Kinase Pathways -- NFkappaB -- Nitric oxide -- Nuclear Receptor Signaling in the Control of Inflammation -- Pathogen-Associated Molecular Patterns (PAMPs) -- Pentraxins -- Prostanoids -- Protease-Activated Receptors -- Reactive oxygen species -- Substance P in Inflammation -- TGF-b superfamily cytokine MIC-1/GDF15 in health and inflammatory diseases -- TNFalpha -- Type I Interferons
    Abstract: Inflammation has become one of the most exciting and rewarding areas of medical research. Recent years have seen a revolution in our understanding of how blood and tissue cells interact and of the intracellular mechanisms controlling their activation. This has revealed the underlying inflammatory pathology of many diseases and provided multiple new targets for anti-inflammatory and immunomodulatory therapy. The Encyclopedia of Inflammatory Diseases will cover the following areas: ℗ ℗ Inflammatory Processes and Cells Inflammatory Diseases Mediators of Inflammation Pharmacology of Inflammation Since inflammatory diseases and their therapy cover a broad range of scientific and medical fields, the encyclopedia will be co-edited by four international experts, a clinician and three researchers from the disciplines of immunology, biochemistry, and pharmacology, in this way providing students, basic and clinical scientists and practitioners in academia, hospitals and industry with valuable interlinked information. This living project will serve as a reliable and comprehensive data pool for everybody working in inflammation research. Owing to its dynamic nature, it will grow with time and future editions, becoming an indispensible source of information for academia, clinical practitioners and industry
    Pages: 218 illus., 147 illus. in color. eReference. : online resource.
    ISBN: 9783764385507
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  • 4
    ISSN: 1420-908X
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Abstract The pannus of rheumatoid arthritis is a proliferating mass of various cells and tissues, including follicles of lymphoid cells, and is the major agent of destruction of the surrounding structural tissues. Infiltrating lymphocytes are thought to be the prime stimuli for the growth of the pannus, through the secretion of lymphokines. In contrast to this high turnover, destructive rheumatoid granuloma, most commonly used experimental granuloma models are of the non-immune, low turnover variety and thus give a rather poor reflection of clinical conditions. A model has been developed in our laboratory in which lymph node cells are removed from Freund's complete adjuvant sensitized rats and injected into sponge implants in normal syngeneic recipient animals, leading to enhancement of granuloma formation. This response has been characterized and shown to be related to lymphocyte activation and to the sensitivity of rats to polyarthritis induction. The lymphocyte transfer model may be useful in studying the action of drugs on lymphocyte-mediated chronic inflammatory responses and the principle of lymphocyte transfer may be applied to other hypersensitivity granuloma models.
    Type of Medium: Electronic Resource
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  • 5
    ISSN: 1420-908X
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Type of Medium: Electronic Resource
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  • 6
    Electronic Resource
    Electronic Resource
    Springer
    Inflammation research 40 (1993), S. iii 
    ISSN: 1420-908X
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Type of Medium: Electronic Resource
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  • 7
    Electronic Resource
    Electronic Resource
    Springer
    Inflammation research 44 (1995), S. 227-227 
    ISSN: 1420-908X
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Type of Medium: Electronic Resource
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  • 8
    ISSN: 1420-908X
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Type of Medium: Electronic Resource
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  • 9
    Electronic Resource
    Electronic Resource
    Springer
    Inflammation research 18 (1986), S. 560-562 
    ISSN: 1420-908X
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Type of Medium: Electronic Resource
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  • 10
    ISSN: 1420-908X
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Type of Medium: Electronic Resource
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