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  • 1
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    Heidelberg : Spektrum Akad. Verl.
    Call number: QH442:153
    Keywords: Genome, Human
    Notes: Transl. of: The human genome.
    Pages: 235 p.
    ISBN: 3-86025-200-3
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    QH442:153 available
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  • 2
    Keywords: Human molecular genetics ; Molecular Biology
    Pages: viii, 151 p. : ill.
    ISBN: 1859961592
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  • 3
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    Oxford : BIOS Scientific Publ.
    Call number: 09-NW:408
    Keywords: Genom / Mensch ; Evolution / Genetik
    Pages: xi, 306 p.
    ISBN: 1-859960-95-2
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    09-NW:408 departmental collection or stack – please contact the library
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  • 4
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    Oxford : BIOS Scientific Publ.
    Call number: H0600:109 ; H0700:50 ; H0900:60 ; C050:2
    Keywords: Human molecular genetics ; Molecular Biology
    Notes: Includes index.
    Pages: xxiii, 576 p. : ill.
    Edition: 2nd ed.
    ISBN: 1-85996-202-5
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    H0600:109 departmental collection or stack – please contact the library
    H0700:50 departmental collection or stack – please contact the library
    H0900:60 departmental collection or stack – please contact the library
    C050:2 departmental collection or stack – please contact the library
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  • 5
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    New York : Garland Press
    Call number: B060:135
    Keywords: Human molecular genetics ; Molecular Biology
    Pages: xxv, 674 p. : ill.
    Edition: 3rd ed.
    ISBN: 0815341849
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    B060:135 departmental collection or stack – please contact the library
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  • 6
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    New York : Garland Press
    Call number: QH447:199(5)
    Keywords: Human molecular genetics ; Molecular Biology
    Pages: xiii, 770 p. : ill.
    Edition: 4th ed.
    ISBN: 9780815345893
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    QH447:199(5) available
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  • 7
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    New York : Garland Press
    Call number: C050:55
    Keywords: Human molecular genetics ; Molecular Biology
    Pages: xxv, 781 p. : ill.
    Edition: 4th rev. ed.
    ISBN: 978-0-8153-4149-9
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    C050:55 available
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  • 8
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    Heidelberg : Spektrum Akad. Verl.
    Call number: H0600:53 ; H0600:65
    Keywords: Human molecular genetics ; Molecular genetics ; Molecular Biology
    Notes: Translation of: Human molecular genetics.
    Pages: xxxix, 743 p. : ill.
    ISBN: 3-8274-0039-2
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    H0600:53 departmental collection or stack – please contact the library
    H0600:65 departmental collection or stack – please contact the library
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  • 9
    ISSN: 1432-1211
    Source: Springer Online Journal Archives 1860-2000
    Topics: Biology , Medicine
    Notes: Abstract We have used the HLA-C-specific DNA probe pC250 to investigate restriction fragment length polymorphism (RFLP) at the HLA-C locus. Genomic Southern blot hybridization included DNA prepared from a panel of homozygous typing cells representing serological specificities Cw1 to Cw8 and also from samples representing Cw blanks. Although many restriction nucleases failed to reveal any polymorphism, RFLPs were evident with Taq I, Pvu II, Bst XI, Nde 1, and Nci I in addition to the previously reported Eco RI. In the case of Bst XI, a unique RFLP defined a subset of serologically defined Cw blanks. Comparison of RFLP sizes with restriction fragment lengths obtained from the known HLA-Cw3 gene sequence permitted the localization of intragenic C locus RFLLs and the identification of a variable Taq I site in the second intron, a variable Nci I site near the end of the fourth exon, and a variable Pvu lI site in the fifth intron.
    Type of Medium: Electronic Resource
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  • 10
    ISSN: 1432-1203
    Source: Springer Online Journal Archives 1860-2000
    Topics: Biology , Medicine
    Notes: Summary The unknown allele that predisposes to the development of haemochromatosis in man has been localized to the HLA class I region on the short arm of chromosome 6. We have utilized pulsed-field gel electrophoresis in conjunction with probes that map within, or in the vicinity of, this region to search for structural lesions that may further define the disease locus. Using the enzyme Mlu I, fragments that associated specifically with the HLA-A23, A31 and B8 alleles were identified. However, in members of three pedigrees affected by haemochromatosis, and in six unrelated patients with the disorder, no disease-specific differences were detected in the DNA fragments with four restriction enzymes and eight probes when compared with healthy individuals. These data suggest that the lesion responsible for hereditary haemochromatosis lies beyond the resolution of this technique and does not involve large structural deletions or extensive re-arrangements in this highly polymorphic region of the genome.
    Type of Medium: Electronic Resource
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