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  • 1990-1994  (2)
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  • 1
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    German Medical Science GMS Publishing House; Düsseldorf
    In:  61. Jahrestagung der Deutschen Gesellschaft für Neurochirurgie (DGNC) im Rahmen der Neurowoche 2010; 20100921-20100925; Mannheim; DOCP1720 /20100916/
    Publication Date: 2010-09-17
    Keywords: ddc: 610
    Language: English
    Type: conferenceObject
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  • 2
    ISSN: 1432-0630
    Keywords: 78.40 ; 78.60 ; 81.20
    Source: Springer Online Journal Archives 1860-2000
    Topics: Mechanical Engineering, Materials Science, Production Engineering, Mining and Metallurgy, Traffic Engineering, Precision Mechanics , Physics
    Notes: Abstract The preparation of GdVO4:Bi3+ ceramics is indicated. Bismuth shows a strong tendency to evaporate during the sintering process. Time-resolved emission spectroscopy shows for sufficiently low Bi3+ concentrations subsequently: blue VO 4 3− emission with a decay time corresponding to the transfer rate (106 s−1), yellow VO 4 3− −Bi3+ emission, rare-earth impurity emission and VO 4 3− −Bi3+ afterglow.
    Type of Medium: Electronic Resource
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  • 3
    ISSN: 1573-7241
    Keywords: cold pressor test ; neuropeptide-Y ; nifedipine ; noradrenaline ; Raynaud's phenomenon
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Summary The effect of a standardized cold pressor test on circulating noradrenaline and neuropeptide-Y-like immunoreactivity was investigated in 12 women with primary Raynaud's phenomenon and 12 healthy female controls before and after 2 weeks of treatment with the calcium antagonist, nifedipine. Measurement before treatment showed significant increase during the cold pressor test on circulating noradrenaline in both the primary Raynaud's phenomenon group and in the control group (from 0.29±0.15 ng/ml to 0.33±0.16 ng/ml, p〈0.05, and from 0.21±0.14 ng/ml to 0.29±0.16 ng/ml, p〈0.005, respectively). However, treatment with nifedipine resulted in significantly increased levels of circulating noradrenaline during the cold pressor test only in the control group (from 0.43±0.21 ng/ml to 0.50±0.20 ng/ml, p〈0.01). Plasma concentrations of neuropeptide-Y-like immunoreactivity were unchanged by the standardized cold pressor test, whether performed before or during nifedipine treatment in both groups. Nifedipine treatment per se significantly increased circulating noradrenaline in both the primary Raynaud's phenomenon patient group and in the control group (from 0.29±0.15 to 0.49±0.13 and 0.21±0.14 to 0.43±0.21 ng/ml, respectively, p〈0.001). Similarly, the circulating neuropeptide-Y-like immunoreactivity significantly increased in both the primary Raynaud's phenomenon group and in the control group (from 105±21 to 137±19 pmol/l and 107±17 to 147±13 pmol/l, respectively, p〈0.001). No significant differences were seen between the two groups, and the increased levels of circulating noradrenaline and neuropeptide-Y-like immunoreactivity were maintained during the entire test period in both groups. The standardized cold pressor test increased circulating noradrenaline significantly in both study groups before treatment with nifedipine. On the other hand, nifedipine moderately increased circulating noradrenaline during the cold pressor test in the primary Raynaud's phenomenon patient group. Besides these findings, this study demonstrates that treatment with nifedipine, a drug widely used in cardiovascular disease, increases circulating levels of noradrenaline and neuropeptide-Y-like immunoreactivity in normotensive women.
    Type of Medium: Electronic Resource
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