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  • 1
    Electronic Resource
    Electronic Resource
    Springer
    Journal of molecular medicine 52 (1974), S. 728-735 
    ISSN: 1432-1440
    Keywords: Muscle blood flow ; 133-Xenon-clearance ; exercise ; upper arm ; forearm ; thumb ; age ; sex ; Muskeldurchblutung ; Belastung ; 133-Xenon-Clearance ; Ober- und Unterarm ; Daumen ; Alter ; Geschlecht ; Seitenunterschiede
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Description / Table of Contents: Zusammenfassung Bei 65 Personen ohne arterielle Zirkulationsstörungen wurde die Muskeldurchblutung der Arme mit der 133-Xenon Clearance in Ruhe, bei Belastung und während der anschließenden reaktiven Hyperämie bestimmt. Im Gesamtkollektiv und bei Männern fand sich mit zunehmendem Alter eine Durchblutungsabnahme. Signifikante Geschlechts-oder Seitenunterschiede waren dagegen nicht festzustellen. Die vergleichende Messung ergab am Oberarm eine signifikante höhere Durchblutung bei Belastung und während der anschließenden reaktiven Hyperämie, als am Unterarm. Die Werte am Unterarm und am Daumenballen unterschieden sich dagegen nicht signifikant.
    Notes: Summary Muscle blood flow (MBF) was determined by 133-Xenonclearance at rest, during exercise and subsequent reactive hyperemia in 65 persons free of vascular symptoms. MBF decreased significantly with increasing age in the whole group and in males. No significant differences were found between males and females, or between the right and the left side. MBF was significantly higher on the upper arm, than on the forearms whereas no differences existed between forearm and thumb.
    Type of Medium: Electronic Resource
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  • 2
    Electronic Resource
    Electronic Resource
    Springer
    Apoptosis 2 (1997), S. 265-282 
    ISSN: 1573-675X
    Keywords: Apoptosis ; cell cycle ; clonogenicity ; p53 ; radiation
    Source: Springer Online Journal Archives 1860-2000
    Topics: Biology , Medicine
    Notes: Abstract Ionizing radiation can be an effective inducer of apoptosis and studies of many aspects of the pathways and mechanisms involved in this apoptosis induction have been published. This review stresses two aspects: the relationship between apoptosis and loss of clonogenic ability in irradiated cells and the time course for the appearance of apoptosis after radiation exposure. Although it was initially assumed that apoptosis occurred relatively quickly (within hours) after irradiation, evidence is presented and discussed here showing that apoptosis can occur at long times after irradiation (out to 20 days) in some cell types. This late, or delayed, apoptosis occurs after the cells have divided once or several times. The impact of delayed apoptosis on loss of clonogenicity after irradiation remains unclear. It seems likely that in some cell types, e.g., fibroblasts, the occurrence of late apoptosis is minimal and may have little impact on long term cell survival of the population, but in at least one instance, with a cell line of hematopoietic origin, it appears that late apoptosis can account for all the loss of clonogenicity in irradiated cells. The role of p53 in radiation-induced apoptosis is also discussed, with data presented showing that both p53-dependent and independent pathways for radiation-induced apoptosis exist, depending on the cell type.
    Type of Medium: Electronic Resource
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