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  • 1
    ISSN: 1432-0428
    Keywords: Microscopic haematuria ; persistent proteinuria ; diabetic glomerulosclerosis ; non-diabetic glomerulopathy ; Type 1 diabetes
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Summary The prevalence and causes of microscopic haematuria were examined in all Type 1 (insulin-dependent) diabetic patients with persistent proteinuria (diabetes duration ⩾5 years) attending the outpatient clinic at Hvidöre Hospital during 1985. One hundred eighty-four patients (69F/115M) out of 1024 Type 1 patients had persistent proteinuria (18%). Microscopic haematuria was defined as ⩾3 erythrocytes per high power field in two or more sterile urine samples. Twenty-three Type 1 patients with persistent proteinuria (7F/16M, aged 35.4±13 years) had microscopic haematuria (12.5%). No significant changes were found between the group with and without microscopic haematuria: blood pressure 148/89±22/11 versus 145/91±20/11 mmHg, duration of diabetes when persistent albuminuria occurred 17±8 versus 20±10 years, serum creatinine 99±24 versus 98±31 μmol/l, simplex retinopathy 61 versus 54%, proliferative retinopathy 39 versus 42%, and no signs of retinopathy 0 versus 4%. Kidney biopsy was performed in 13 out of the 23 patients with microscopic haematuria. Diabetic glomerulosclerosis was present in all 13 patients, but 9 patients had a non-diabetic renal disease superimposed (mesangioproliferative glomerulonephritis (n=5), membranous glomerulonephritis (n=3) and sarcoidosis (n=1). Microscopic haematuria is a rare finding, frequently reflecting superimposed non-diabetic glomerulopathies, in Type 1 diabetic patients with diabetic nephropathy and well preserved kidney function.
    Type of Medium: Electronic Resource
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  • 2
    ISSN: 1591-9528
    Keywords: Shock ; Blood Cytology ; Circulatory failure ; Shock, experimental ; Bone marrow, Cytology ; Schock ; experimenteller Schock ; Blut-Cytologie ; Knochenmarks-Cytologie
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Description / Table of Contents: Zusammenfassung 1. In Untersuchungen an Leukocytenkonzentraten des peripheren Blutes von 40 Patienten mit klinisch gesichertem Schock und von 20 Kontrollpersonen ohne Schockzeichen konnte gezeigt werden, daß Plasmoblasten, Proplasmazellen, Plasmazellen, Promyelocyten, Myelocyten und Erythroblasten bei den Schockpatienten statistisch signifikant häufiger vorkamen als in der Kontrollgruppe. 2. Weiterhin wurden Zellkonzentrate aus dem Blut und aus den Nierenperfusaten von 15 Kaninchen mit experimentellem Entblutungsschock und von 10 Kontrolltieren cytologisch untersucht. Folgende Zellformen wurden in der Schockgruppe statistisch signifikant häufiger angetroffen: in den Blutkonzentraten Myelocyten und Plasmoblasten; in den Nierenperfusaten Myelocyten, Proplasmazellen und Plasmazellen. 3. Die Gesamtzahl kernhaltiger Zellen (einschließlich Lymphocyten und segmentkerniger Granulocyten) war in den Ausstrichpräparaten der Konzentrate aus Blut und Nierenperfusat der Schocktiere statistisch signifikant höher als in den entsprechenden Präparaten der Kontrolltiere. 4. Die Ergebnisse zeigen, daß Schockzustände mit einer vermehrten Ausschwemmung der genannten Zellen in das periphere Blut einhergehen, und tragen somit dazu bei, das Auftreten intravasaler Zellansammlungen in menschlichen Schocknieren zu erklären.
    Notes: Summary 1. Leucocyte concentrates of peripheral blood from 40 patients with shock and 20 control persons without clinical symptoms of shock were investigated for the presence of immature blood and bone marrow cells. The number of cases containing plasmoblasts, proplasmocytes, plasmocytes, promyelocytes, myelocytes, and erythroblasts, was significantly higher in the shock group. 2. Leucocyte concentrates were also investigated from the blood and from the kidney perfusion fluid of 15 rabbits with experimental hemorrhagic shock and 10 control animals. The blood concentrates of the shock group contained significantly more myelocytes and plasmoblasts, the kidney perfusates significantly more myelocytes, proplasmocytes, and plasmocytes. 3. The number of nucleated cells including lymphocytes and polymorphonuclear leucocytes was significantly higher in the smears of the leucocyte concentrates from blood and kidney perfusates within the shock group compared to the control group. 4. It may be concluded from the present results that circulatory failure is accompanied by increased delivery of immature cells into the blood. The cell accumulations in the renal blood vessels following shock may be explained by this mechanism.
    Type of Medium: Electronic Resource
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